By Narayan Chakrabarty
The so much noted metalloid within the glossy international, arsenic impacts the liver, kidney, and lungs; ends up in cardiovascular illnesses, melanoma, and diabetes; and should reason blindness with long-time publicity. With evidently taking place arsenic boosted via mining and different business tactics contaminating soil and ingesting water, arsenic toxicity is an important problem to well-being execs and scientists around the globe. Arsenic Toxicity: Prevention and Treatment reports present knowing of arsenic poisoning and the well-being results because of publicity.
The ebook paints a vibrant photo of the resources of arsenic toxicity together with floor water; foodstuff akin to rice, vegetables and fruit, fish, and chook in addition to occupational exposures from industries utilizing inorganic arsenic such as glass construction, non-ferrous alloy, wooden protection, and semiconductor production units. The text details the well-being dangers of arsenic toxicity after which examines elimination, ordinarily from soil and water, highlighting green bioremediation options. It discusses classical and smooth therapy tools for arsenic toxicity, emphasizing using nutraceuticals and practical foods.
With its concentrate on the remediation of arsenic toxicity utilizing nutraceutical and practical meals, the ebook offers a distinct source for combatting this international scourge. It offers concepts for protecting arsenic toxicity clearly with out inflicting any extra antagonistic results.
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Extra info for Arsenic Toxicity: Prevention and Treatment
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A couple of studies suggested the association between histone phosphorylation and arsenic carcinogenesis. Li et al. suggest that arsenic trioxide induces histone H3 phosphoacetylation at the caspase-10 gene, which may play a role in apoptosis contributing to the anticancer effects of arsenic trioxide on acute promyelocytic leukemia . One year later, they showed that arsenite stimulates histone H3 phosphoacetylation of proto-oncogenes c-fos and c-jun chromatin in lung fibroblasts . All these studies clearly demonstrated that histone modification is dysregulated by arsenic exposure, but further work is required to understand the overall effects of altered histone modification on arsenic-induced toxicity or carcinogenesis.